con-a hrp Search Results


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EY Laboratories hrp-conjugated con a
Hrp Conjugated Con A, supplied by EY Laboratories, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Seikagaku corporation horseradish peroxidase (hrp) conjugated lectins cona and wga
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US Biological Life Sciences concanavalin a (cona) hrp conjugate
(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry <t>(ConA</t> lectin, bottom graphs).
Concanavalin A (Cona) Hrp Conjugate, supplied by US Biological Life Sciences, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Bangalore Genei India Pvt Ltd hrp conjugated cona
(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry <t>(ConA</t> lectin, bottom graphs).
Hrp Conjugated Cona, supplied by Bangalore Genei India Pvt Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Jackson Laboratory hrp-concanavalin a (cona) antibody
(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry <t>(ConA</t> lectin, bottom graphs).
Hrp Concanavalin A (Cona) Antibody, supplied by Jackson Laboratory, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Seikagaku corporation cona-hrp
(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry <t>(ConA</t> lectin, bottom graphs).
Cona Hrp, supplied by Seikagaku corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Nanoprobes Inc cona-popcorn-hrp nanoprobes
(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry <t>(ConA</t> lectin, bottom graphs).
Cona Popcorn Hrp Nanoprobes, supplied by Nanoprobes Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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EY Laboratories con-a hrp
(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry <t>(ConA</t> lectin, bottom graphs).
Con A Hrp, supplied by EY Laboratories, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Seikagaku corporation hrp-labeled con a
(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry <t>(ConA</t> lectin, bottom graphs).
Hrp Labeled Con A, supplied by Seikagaku corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Alpha Diagnostics cona-hrp antibody
(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry <t>(ConA</t> lectin, bottom graphs).
Cona Hrp Antibody, supplied by Alpha Diagnostics, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Seikagaku corporation hrp-cona 4
(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry <t>(ConA</t> lectin, bottom graphs).
Hrp Cona 4, supplied by Seikagaku corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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EY Laboratories con a–horseradish peroxidase (hrp)
(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry <t>(ConA</t> lectin, bottom graphs).
Con A–Horseradish Peroxidase (Hrp), supplied by EY Laboratories, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


(a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry (ConA lectin, bottom graphs).

Journal: Scientific Reports

Article Title: TUSC3 Loss Alters the ER Stress Response and Accelerates Prostate Cancer Growth in vivo

doi: 10.1038/srep03739

Figure Lengend Snippet: (a) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were evaluated using transmission electron microscopy in ultrathin sections. Only cells with intact plasma membrane, nucleus, nucleolus, mitochondria, ER and Golgi apparatus were included into the analysis. We examined 20 cells of each cell type. Arrows indicate branching or dilatation of ER cisternae. Scale bars correspond to 2 μm. TUSC3 silencing promotes prostate cancer xenograft growth. (b) shTUSC3 and control (scrambled shRNA) PC3 and DU145 cells, respectively, were injected into the flanks of 6–8-week old male nude mice. Tumor growth was monitored and is shown as mean tumor size (+/− SEM). (c) Representative tumor formed by the TUSC3 silenced PC3 xenograft in the right flank of a Foxn1 nu/nu mouse (top left). Sustained downregulation of TUSC3 in mouse tumors was analyzed by qRT-PCR (top right) and immunohistochemistry (TUSC3 IHC, middle graphs). Major differences in N-glycosylation in mouse tumors were not identified by Concanavalin A lectin histochemistry (ConA lectin, bottom graphs).

Article Snippet: Lectin histochemistry and cytochemistry was performed using Concanavalin A (ConA) HRP conjugate in a concentration of 1:40 in PBS (USBiological, Swampscott, MA, USA) overnight at 4°C, followed by 2xPBS washes and visualized with Dako Liquid DAB + Substrate-Chromogen System (K3468).

Techniques: Control, shRNA, Transmission Assay, Electron Microscopy, Clinical Proteomics, Membrane, Injection, Quantitative RT-PCR, Immunohistochemistry, Glycoproteomics